ACS, ischemic heart disease
A good strain in ACS = good prognosis & good survival in patients with normal EF, and therefore, limited additional prognostic value
Ischemic CMP & non-ischemic CMP GLS > -6,95%, there is a drastic risk increase for CV-events
< -6,95% in ischemic CMP, higher risk than non-ischemic (independent of EF) for CV-events
Dispersion >60ms ➜ substrate for arrhythmia (ischemic CMP)
PSS & ESL ➜ the more ischemia (reduction of transmural bloodflow), the higher it is
More PSS, better recovery after CAG
Ischemic Memory via PSS & ESL
GLS >-12% as severely reduced strain in CAD, -15 to -16% as risk marker, PSI >10% in borderline Strain predictor for mortality
TakoTsubo
More positive strain compared with myocardial infarction (TC -11%, ACS -14%)
Segmental strain ratio (SSR) can be calculated – The future: RASr / FWS global
Pulmonary embolism
FWS worsening as a marker of bad prognosis in PE (-14,4% ±7,2%)
In pulmonary hypertension a FWS with -15% & a GS with -15,3% as cut-off for worse prognsis
RASr reduction in in acute PE
FWS — basal & mid reduction in PE compared to apical values („Strain“ McConnell Sign)
RV-PA coupling, pulmonary edema
RV-PA coupling in systemic sclerosis — PHT & adverse outcome (TAPSE / PASP <0,4)
- FWS global / PASP ratio <0,46 %/mmHg
- FWS basal / PASP ratio <0,54 %/mmHg
- Future RASr / FWS global
LASr with 16,2% as a predictor for pulmonary edema in heart failure patients
In pulmonary edema LASr decreased with 9,2%; vs 21,5% in HF without pulmonary edema
Worse LA Strain ➜ more pulmonary edema
LASr as a marker of LV-failure and LA-pressure rise until pulmonary edema as a life-threatening condition in heart failure
